Radang: Perbedaan antara revisi

|Cleaves to produce  ''C3a''  and  ''C3b''. C3a stimulates histamine release by mast cells, thereby producing vasodilation. C3b is able to bind to bacterial cell walls and act as an  opsonin, which marks the invader as a target for  phagocytosis.

|Stimulates histamine release by mast cells, thereby producing vasodilation. It is also able to act as a  chemoattractant  to direct cells via chemotaxis to the site of inflammation.

|A protein that circulates inactively, until activated by collagen, platelets, or exposed  basement membranes  via  conformational change. When activated, it in turn is able to activate three plasma systems involved in inflammation: the kinin system, fibrinolysis system, and coagulation system.

|A complex of the complement proteins  C5b,  C6,  C7,  C8, and multiple units of  C9. The combination and activation of this range of complement proteins forms the''membrane attack complex'', which is able to insert into bacterial cell walls and causes cell lysis with ensuing death.

|Cleaves the soluble plasma protein  fibrinogen  to produce insoluble  fibrin, which aggregates to form a  blood clot. Thrombin can also bind to cells via the  PAR1receptor to trigger several other inflammatory responses, such as production of  chemokines  and  nitric oxide.