Protein Kinase B: Perbedaan antara revisi
Konten dihapus Konten ditambahkan
Luckas-bot (bicara | kontrib) k r2.7.1) (bot Menambah: de, es, fr, ru, zh |
dev |
||
Baris 16:
| accessdate = 2011-07-13
| work = Departments of ‡Molecular Oncology, Thoracic Oncology, and Drug Discovery, H. Lee Moffitt Cancer Center and Research Institute, Tianjin Key Laboratory of Lung Cancer Metastasis and Tumor Microenvironment, Lung Cancer Institute, Tianjin Medical University General Hospital; Donghwa Kim, Mei Sun, Lili He, Qing-Hua Zhou, Jun Chen, Xia-Meng Sun, Gerold Bepler, Said M. Sebti, dan Jin Q. Cheng
}}</ref> Akt juga teraktivasi oleh [[obat|obat-obatan]], [[toksin]], [[radikal bebas]] seperti [[litium]], [[asam valproat]], ''clostridium difficile toxin'', ''tert-butylhydroquinone'', [[oksigen]] singlet, dan [[nitrogen monoksida]].<ref>{{en}}{{cite web
| url = http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1945105
| title = Protein kinase B/Akt modulates nephrotoxicant-induced necrosis in renal cells
| accessdate = 2011-07-14
| work = Department of Pharmaceutical Sciences, College of Pharmacy, University of Arkansas for Medical Sciences; Department of Pharmacology and Toxicology, University of Arkansas for Medical Sciences; Zabeena P. Shaik, E. Kim Fifer, dan Grażyna Nowak
}}</ref> Pada [[sel saraf]], [[miosit]] [[jantung]], [[sel epitelial]] [[paru]], aktivasi dapat diinduksi oleh [[simtoma]] seperti [[hipoksia]], reoksigenasi, [[iskemia]]-reperfusi dan [[stres oksidatif]].
== Rujukan ==
| |||