Sistem imun bawaan: Perbedaan antara revisi
Konten dihapus Konten ditambahkan
Baris 175:
| accessdate = 2010-03-14
| work = Dipartimento di Medicina Sperimentale, Università degli Studi di Genova, Istituto Nazionale per la Ricerca sul Cancro, et al, Alessandro Moretta, Cristina Bottino, Massimo Vitale, et al.
}}</ref> Molekul MHC-I dari sel target dipindai oleh pencerap ''killer-inhibitory'' sel NK. Virus, stres, transformasi malignan maupun sel tumor, sel terinfeksi virus akan mempunyai molekul MHC-I yang berbeda, sehingga sel NK akan melakukan [[apoptosis]] terhadap sel tersebut.
Sel NK kemudian mensekresi protei seperti [[perforin]], kemokina dan [[enzim]] proteolitik, [[granzime]]. Granzime yang dilepaskan akan
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The NK cell then releases pore-forming proteins called perforins, proteolytic enzymes called granzymes, and chemokines. Granzymes pass through the pores and activate the enzymes that lead to apoptosis of the infected cell by means of destruction of its structural cytoskeleton proteins and by chromosomal degradation. As a result, the cell breaks into fragments that are subsequently removed by phagocytes (see Fig. 5). Perforins can also sometimes result in cell lysis.
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==Pertahanan keenam==
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